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<div class=Section1>

<p class=MsoNormal>&nbsp; </p>

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  <p class=MsoNormal><o:p>&nbsp;</o:p></p>
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  <td style='padding:0in 0in 0in 0in'>
  <p class=MsoNormal><b><span style='font-family:Arial'>The Dr. Ewan Cameron
  Vitamin C Treatment Protocol for Cancer</span></b></p>
  </td>
 </tr>
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  <td width=132 valign=top style='width:99.0pt;padding:0in 0in 0in 0in'>
  <p class=MsoNormal><span style='color:red'>Cameron Protocol</span> <br>
  <a href="index.html">Home</a></p>
  </td>
  <td width=18 style='width:13.5pt;padding:0in 0in 0in 0in'>
  <p class=MsoNormal><o:p>&nbsp;</o:p></p>
  </td>
  <td valign=top style='padding:0in 0in 0in 0in'>
  <p class=MsoNormal><i><span style='font-family:Arial'>&quot;It has been known
  for many years that cancer patients have depressed circulating, cellular, and
  tissue ascorbate reserves, and ascorbate (vitamin C) is involved in many
  aspects of host resistance to cancer.&quot;&nbsp;</span></i> </p>
  <p><b style='mso-bidi-font-weight:normal'><span style='font-family:Arial'>PROTOCOL
  FOR THE USE OF INTRAVENOUS VITAMIN C IN THE TREATMENT OF CANCER (1986)</span>
  <o:p></o:p></b></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>by Ewan Cameron, MD,
  FRCS, Medical Director, Linus Pauling Institute of Science and Medicine.
  Formerly Chief of Surgery, Vale of <st1:PlaceName w:st="on">Leven</st1:PlaceName>
  <st1:PlaceType w:st="on">Hospital</st1:PlaceType>, <st1:place w:st="on"><st1:City
   w:st="on">Lochlomondside</st1:City>, <st1:PostalCode w:st="on">G83 OUA</st1:PostalCode>,
   <st1:country-region w:st="on">Scotland</st1:country-region></st1:place></span><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>Reprinted with permission
  of the Linus Pauling Institute, <st1:place w:st="on"><st1:PlaceName w:st="on">Oregon</st1:PlaceName>
   <st1:PlaceType w:st="on">State</st1:PlaceType> <st1:PlaceType w:st="on">University</st1:PlaceType></st1:place></span><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>(Note: Within this paper,
  Dr. Cameron cited 38 references, and recommended 11 additional sources. The
  complete bibliography is posted at <span style='mso-bidi-font-weight:bold'><a
  href="http://www.doctoryourself.com/biblio_cameron.html">http://www.doctoryourself.com/biblio_cameron.html</a></span></span><span
  style='font-size:11.0pt'> ) <o:p></o:p></span></p>
  <p><i><span style='font-size:11.0pt;font-family:Arial'>Important Note: This
  paper is designed specifically for, and addressed specifically to, licensed
  medical physicians.</span></i><span style='font-size:11.0pt'> <br>
  &nbsp; <o:p></o:p></span></p>
  <p><b><span style='font-size:11.0pt;font-family:Arial'>MEDICO-LEGAL
  PRECAUTIONS</span></b><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>More than one thousand
  (as of 1986) cancer patients have been treated by supplemental ascorbate
  (vitamin C) in a few canters in <st1:country-region w:st="on">Scotland</st1:country-region>
  and <st1:place w:st="on"><st1:country-region w:st="on">Japan</st1:country-region></st1:place>.
  In addition, it has been reliably estimated that at least 100,000 cancer
  patients in the <st1:place w:st="on"><st1:country-region w:st="on">United
    States</st1:country-region></st1:place> are currently ingesting ascorbates
  with or without the tacit consent and knowledge of their physicians.
  Nevertheless this form of treatment must still be considered experimental and
  unproven. Before initiating treatment, the physician must obtain signed
  informed consent stressing this point. The administration of ascorbate either
  orally or intravenously is not illegal anywhere in the <st1:country-region
  w:st="on">United States</st1:country-region> or <st1:place w:st="on"><st1:country-region
   w:st="on">Canada</st1:country-region></st1:place>.</span><span
  style='font-size:11.0pt'> <br>
  &nbsp; <o:p></o:p></span></p>
  <p><b><span style='font-size:11.0pt;font-family:Arial'>TREATMENT RATIONALE</span></b><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>The reasons for the
  expectation that supplemental ascorbate should be of some benefit to all
  cancer patients have been given in detail elsewhere. To summarize, it has
  been known for many years that cancer patients have depressed circulating,
  cellular, and tissue ascorbate reserves, and ascorbate (vitamin C) is
  involved in many aspects of host resistance to cancer. Moreover, <b>ascorbate
  administered in pharmacological doses enhances many parameters of the immune
  response to levels far above the somewhat arbitrary &quot;normal&quot; range,
  including cell-mediated immunity and the endogenous production of interferon.
  There is also some laboratory evidence behind the strong clinical impression
  that in certain situations attainable concentrations of ascorbate may have a
  selective cytotoxic effect on malignant cells while being harmless to normal
  cells</b>, resulting in cancer regression in rare instances.</span><span
  style='font-size:11.0pt'> <br>
  &nbsp; <o:p></o:p></span></p>
  <p><b><span style='font-size:11.0pt;font-family:Arial'>THE CONTROVERSY</span></b><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>Despite the above, it is
  understandably difficult to believe that a simple cheap and safe substance
  such as vitamin C could have any possible value against such a bafflingly
  complex disease as cancer. Such reasonable skepticism was seemingly confirmed
  by the publication of <b>two negative trials carried out by respected
  investigators at the Mayo Clinic. However, careful reading of these two
  papers shows that these much publicized results were far from conclusive.
  Both trials relied upon oral medication only</b>, and in the form of 20
  ascorbate capsules to be swallowed per day, compared to controls swallowing
  20 capsules of an easily recognized placebo. In the first trial 127
  far-advanced patients suffering from miscellaneous cancers were studied,
  almost all of whom had already been unsuccessfully treated by chemotherapy or
  radiation therapy and often both. Such immune-compromised patients would be
  the least likely to derive any benefit from vitamin C. Furthermore no checks
  were carried out to ensure that controls were not self-medicating with this
  freely available substance.&nbsp;</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>The second trial was even
  more inconclusive. Ascorbate or placebo was administered to 100 patients with
  inoperable colon cancer, abruptly discontinued when tumor progression was
  detected at a median time of 10 weeks, then replaced in the majority by
  chemotherapy. From the published data <b>in the second paper it is clear that
  the few controls tested were also self-medicating with vitamin C</b>. Thus
  the ascorbate treatment of cancers remains unproven and experimental, but by
  no means disproved.</span><span style='font-size:11.0pt'> <br>
  &nbsp; <o:p></o:p></span></p>
  <p><b><span style='font-size:11.0pt;font-family:Arial'>TREATMENT CHOICE</span></b><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>Cancer treatment must
  never be regarded as an alternative to proven conventional methods of cancer treatment,
  but instead as a supportive measure employed alongside whatever standard
  method is indicated. There is some evidence of synergism. Thus ascorbate has
  long been known to promote healing, and should accelerate convalescence from
  major cancer surgery. Ascorbic acid and its metabolite dehydroascorbic acid
  radiosensitize hypoxic tumor cells with the potential to enhance the
  therapeutic effects of radiation. Ascorbate enhances the chemotherapeutic
  activity of some chemotherapeutic drugs, including adriamycin, while there is
  considerable anecdotal evidence that ascorbate diminishes to a certain extent
  the unpleasant side-effects of cytotoxic chemotherapy without apparently
  interfering with the effectiveness of such drugs, although the latter point
  has not been completely investigated.</span><span style='font-size:11.0pt'> <br>
  &nbsp; <o:p></o:p></span></p>
  <p><b><span style='font-size:11.0pt;font-family:Arial'>BASELINE WORKUP</span></b><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>As with any cancer
  treatment, it is important to establish certain baseline data before
  commencing treatment, in order to monitor future therapeutic response. Such
  baseline data will naturally vary depending on the type and extent of the
  particular cancer being treated but will include patient's weight,
  performance on the Karnovsky scale, full hematological profile, SMAC-16
  biochemical profile, measurement of serum tumor marking proteins if present,
  and clinical and radiological (including CAT-scan and NMR imaging if
  appropriate) measurement of the extent of tumor load.</span><span
  style='font-size:11.0pt'> <br>
  &nbsp; <o:p></o:p></span></p>
  <p><b><span style='font-size:11.0pt;font-family:Arial'>PRINCIPLE OF TREATMENT</span></b><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>Clinical experience
  indicates that the best responses are obtained if one can maintain a
  continuous high plasma ascorbate level. Ascorbate, however administered, is
  rapidly excreted in the urine, so that administration should be continuous or
  at very frequent intervals. Furthermore, exposure to high circulating levels
  of ascorbate induces over-activity of certain hepatic enzymes concerned with
  its degradation and metabolism. These enzymes persist for some time after
  sudden cessation of high intakes, resulting in depletion of circulating
  levels of ascorbate to well below normal unsupplemented values. This is known
  as the rebound effect. It causes a sharp decrease in immunocompetence and
  must be avoided in the cancer patient. Clinical experience has shown that the
  best responses are observed when vitamin C is administered intravenously, so
  insuring a high plasma level. However, because long-term continuous
  intravenous administration is impractical, we recommend an initial
  intravenous course of ten days duration, followed by continuous maintenance
  oral regimen. If the patient's condition deteriorates, further intravenous
  &quot;booster&quot; courses are recommended.</span><span style='font-size:
  11.0pt'> <br>
  &nbsp; <o:p></o:p></span></p>
  <p><b><span style='font-size:11.0pt;font-family:Arial'>INTRAVENOUS VITAMIN C</span></b><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>Vitamin C intravenously
  can be given by intermittent injection, but this is not recommended,
  particularly if the intervals between injections extend to several days.
  Because of the rebound effect, such administration would produce a sawtooth
  plasma ascorbate profile with abnormally low levels in the troughs just prior
  to the next injection. We recommend continuous administration via slow drip
  infusion.</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>It is recommended that
  the patient be hospitalized for initial evaluation and commencing the
  intravenous regimen. Intravenous vitamin C can cause troublesome chemical
  phlebitis if injected directly into a superficial vein. Fo0r this reason it
  is recommended that infusion be given via a C.V.P. (Central Venous Pressure,
  or similar) line, with routine A-P portable chest X-ray taken after placement
  to ensure that the catheter tip is correctly positioned in the Superior Vena
  Cava. Alternately a &quot;Hep-Lock&quot; (or similar) cannula may be used in
  a forearm vein with appropriate precautions against clotting.</span><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><b style='mso-bidi-font-weight:normal'><span style='font-size:11.0pt;
  font-family:Arial'>(Additional information on intravenous vitamin C
  administration is found <span style='mso-bidi-font-style:italic'>in<i> A
  Physician's Handbook of Orthomolecular Medicine</i></span>, Roger J.
  Williams, editor; at the website of Robert Cathcart, MD (</span></b><span
  style='font-size:11.0pt;font-family:Arial'><a href="http://www.orthomed.com/">http://www.orthomed.com</a><b
  style='mso-bidi-font-weight:normal'>); in Dr. Cathcart&#8217;s paper entitled
  <i>Preparation of Sodium Ascorbate for IV and IM Use </i><span
  style='mso-bidi-font-style:italic'>posted at </span></b><span
  style='mso-bidi-font-style:italic'><a
  href="http://www.doctoryourself.com/vitciv.html">http://www.doctoryourself.com/vitciv.html</a><b
  style='mso-bidi-font-weight:normal'> </b></span><b style='mso-bidi-font-weight:
  normal'>; and in <i>Clinical Guide to the Use of Vitamin C</i>, by Lendon
  Smith, MD. The full text of Dr. Smith&#8217;s book is posted at</b> </span><span
  style='font-size:9.0pt;font-family:Arial'><a
  href="http://www.seanet.com/%7Ealexs/ascorbate/198x/smith-lh-clinical_guide_1988.htm">http://www.seanet.com/~alexs/ascorbate/198x/smith-lh-clinical_guide_1988.htm</a>
  </span><b style='mso-bidi-font-weight:normal'><span style='font-size:11.0pt;
  font-family:Arial'>. These sources discuss details including needle gauges
  and buffers, among other topics.)</span></b><b style='mso-bidi-font-weight:
  normal'><span style='font-size:11.0pt'> <o:p></o:p></span></b></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>The recommended
  &quot;carrier solution&quot; is Ringer's Lactate Solution, readily available
  in liter packs or flasks, and infused at the steady rate of 2 liters (two
  packs) every 24 hours. Isotonic Dextrose should <b>never</b> be used as the
  carrier solution. Vitamin C for parenteral use comes in sterile ampules of
  sodium ascorbate preferably without preservative. Standard ampules contain
  0.5g (500 milligrams), or as &quot;multiuse&quot; 50 ml ampules containing 25
  grams. The ampules should be added to the infusion packs under full sterile
  conditions in the Hospital Pharmacy or Infusion Fluids Department.</span><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>In patients not
  previously exposed to high levels of ascorbate, a gradual &quot;wind-up&quot;
  dose regimen is recommended, along the following lines:</span><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>Day One to the first
  flask add 0.5g sodium ascorbate</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>to the second 1.0g sodium
  ascorbate</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>to the third 1.5g sodium
  ascorbate</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>to the fourth 2.0g sodium
  ascorbate</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>Day Two to the fifth
  flask add 2.5g sodium ascorbate</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>(continuing at that level
  infuses 10g sodium ascorbate per day)</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>Much higher doses can be
  and have been infused with perfect safety, the limiting factor being sodium
  overload with water retention, and not the ascorbate ion itself. The wind-up
  dosage schedule is not necessary in patients receiving &quot;booster&quot;
  repeat courses.</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>PRECAUTIONS: Standard
  continuous infusion procedure must be followed. It is not necessary to
  monitor serum electrolyte levels except in patients with gross cardiac or
  renal impairment.</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>SIDE-EFFECTS There are
  two recognized side-effects, one very common and relatively harmless, the
  other very rare and highly dangerous. The common side-effect is transient
  fluid retention due to sodium overload resulting in some ankle edema in the
  ambulant and sacral pad edema in those confined to bed. Inpatients with
  cardiac impairment dangerous pulmonary edema may develop and require
  intravenous frusemide for its control.</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>The rare side-effect,
  seen only in patients with highly anaplastic very rapidly growing tumors and
  a heavy tumor load, is the sudden precipitation of widespread tumor necrosis.
  Clinically this is heralded by sudden pain in all tumor deposits, rapid
  swelling of known tumors, tumor hemorrhage, both internal and external,
  hyperpyrexia, severe hypotension,</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>tachycardia and azotemia.
  This very rare complication can be fatal and must be vigorously treated. If
  suspected, the ascorbate infusion should be immediately stopped, and the
  patient treated as for septicemic shock. The patient may require transfer to
  the Intensive Care Unit for close monitoring and support by oxygen, plasma or
  blood, and intravenous steroids. If resuscitation is successful, it will be
  found that any residual tumor has shrunk considerably or even disappeared.
  Although highly dangerous, this reaction might also be termed the best
  possible response to ascorbate treatment of wide-spread cancer.</span><span
  style='font-size:11.0pt'> <br>
  &nbsp; <o:p></o:p></span></p>
  <p><b><span style='font-size:11.0pt;font-family:Arial'>ORAL VITAMIN C</span></b><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>On the day planned to
  terminate intravenous ascorbate, the patient should be commenced on oral
  ascorbate at the same daily intake. The medication should be taken every six
  hours (four times a day), and once commenced should never be abruptly
  discontinued because of the rebound effect. The dose varies between
  individuals in the 10 to 30g a day range. The aim should be to maintain
  plasma ascorbate levels of at least 3mg/dl for effective therapy. Leukocyte
  ascorbate concentration, a technically more difficult estimation to perform,
  is of much less value in predicting response to treatment.</span><span
  style='font-size:11.0pt'> <br>
  &nbsp; <o:p></o:p></span></p>
  <p><b><span style='font-size:11.0pt;font-family:Arial'>RESPONSE TO ASCORBATE
  TREATMENT</span></b><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>As with any other form of
  cancer treatment, therapeutic response will vary between individual patients,
  with paradoxically the best responses seen with individuals with far advanced
  disease. Therapeutic responses may be listed as follows:</span><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>1. No response detectable
  (a very few)</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>2. Some retardation of
  progressive tumor growth (very many)</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>3. Stasis or standstill
  effect (considerable number)</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>4. Tumor regression (a
  few)</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>5. Tumor necrosis (very
  rare)</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>A typical response to
  ascorbate can be described as follows:</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>An improvement in
  well-being, vigor and Karnovsky performance status will be apparent in 5 to 7
  days. Originally thought to be a purely subjective response, it is now
  realized to be objective and due to restoration of endogenous carnitine
  biosynthesis, carnitine being responsible for transporting triglycerides
  across the mitochondrial membrane where they are burned for muscle energy.</span><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>If painful skeletal
  metastases are present, relief of bone pain will occur in about 5 to 7 days, <b>enabling
  opiates to be withdrawn without withdrawal symptoms</b>. Skeletal or
  widespread visceral metastases are associated with increased urinary
  hydroxyproline (UHP) excretion reflecting collagen breakdown. <b>Within 5
  days of commencing ascorbate therapy, a sharp and sustained fall in UHP
  excretion will be noted</b>.</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>Response to treatment
  will also be reflected in a drop in the sedimentation rate and fall in the
  titer of any serum tumor protein markers (CEA etc.) if present.</span><span
  style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>Radiological signs of
  good response include the slow conversion of osteolytic skeletal metastases
  to dense osteosclerotic lesions over a period of months. In favorable cases,
  resorption of malignant pleural effusions and reduction in size of pulmonary
  metastases have been observed.</span><span style='font-size:11.0pt'> <br>
  &nbsp; <o:p></o:p></span></p>
  <p><b><span style='font-size:11.0pt;font-family:Arial'>CONTINUATION OF
  TREATMENT</span></b><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>Oral ascorbate should be
  continued indefinitely and the patient carefully monitored at least every
  month. The usual course of events is that the patient instead of slowly
  declining will enter a plateau of comparative well-being that may continue
  for many months or even years, and then enter an abrupt downhill phase with
  explosive metastases. At the first suspicion of this change, a
  &quot;booster&quot; course of intravenous ascorbate is recommended, even
  though individual responses can never be predicted. Over a period of three
  years, one patient with widespread leiomyosarcoma had six such booster courses
  with clear benefit from each one except the last. Many other patients have
  shown no benefit from even the first booster course, but it is always worth
  trying.</span><span style='font-size:11.0pt'> <br>
  &nbsp; <o:p></o:p></span></p>
  <p><b><span style='font-size:11.0pt;font-family:Arial'>INTRACAVITARY
  ASCORBATE</span></b><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>Isotonic solutions of
  sodium ascorbate (2.97g sodium ascorbate in 100 ml sterile distilled water
  for injection) have been instilled in the peritoneal and pleural cavities
  after paracentesis or thoraocentesis for malignant effusions. Benefit
  appeared to ensue in the sense of reduction in the rate of effusion
  accumulation, although without controls this impression is difficult to
  prove. At least clinical experience has shown that the procedure is quite
  painless and perfectly safe.</span><span style='font-size:11.0pt'> <br>
  &nbsp; <o:p></o:p></span></p>
  <p><b><span style='font-size:11.0pt;font-family:Arial'>KIDNEY
  &quot;DAMAGE&quot;</span></b><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-size:11.0pt;font-family:Arial'>There is a widespread
  belief in the medical profession that large intakes of vitamin C might
  somehow cause kidney damage. This apparently stems from a (1954) study that
  showed that in some individuals (5 percent of those tested) intakes of
  vitamin C above 4 grams per day resulted in a measurable increase in urinary
  oxalate levels with a theoretical risk of oxalate stone formation. Doubts
  have been expressed about the specificity of the assay method used, and
  studies on healthy individuals ingesting large amounts of vitamin C for years
  have shown plasma and urinary oxalate levels well within normal range. No
  instance of this complication has been encountered in over 100 cancer
  patients under the author's care, or in many other patients ingesting
  multigram amounts of ascorbate for many years. The risk of stone formation,
  if it exists at all, must be extremely remote, and more than offset by the
  potential benefit to the cancer patient. (Additional information refuting
  alleged vitamin-caused kidney stones is found in <i>The Vitamin C Connection</i>,
  by Emanuel Cheraskin, MD, et al, and <i>How to Live Longer and Feel Better</i>,
  by Linus Pauling, PhD.)</span><span style='font-size:11.0pt'> <o:p></o:p></span></p>
  <p><span style='font-family:Arial'>(Note: Within this paper, Dr. Cameron
  cited 38 references, and recommended 11 additional sources. The complete
  bibliography is posted at </span><span style='font-size:11.0pt'><a
  href="http://www.doctoryourself.com/biblio_cameron.html"><span
  style='font-family:Arial;mso-bidi-font-weight:bold'>http://www.doctoryourself.com/biblio_cameron.html</span><span
  style='font-family:Arial'>)</span></a></span><span style='font-size:11.0pt;
  font-family:Arial'> <o:p></o:p></span></p>
  <p><b style='mso-bidi-font-weight:normal'><span style='font-size:10.0pt;
  font-family:Arial'>Andrew Saul is the <span class=grame>author of the books <i>FIRE</i></span><i>
  YOUR DOCTOR!</i> How <i>to be Independently Healthy </i>(reader reviews at<i>
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  ) and <i>DOCTOR YOURSELF: Natural Healing that Works.</i> (reviewed at <a
  href="http://www.doctoryourself.com/saulbooks.html">http://www.doctoryourself.com/saulbooks.html</a>
  ) </span><o:p></o:p></b></p>
  <p><b style='mso-bidi-font-weight:normal'><span style='font-size:10.0pt;
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